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INS1 -cell mitochondrial matrix Ca 2+ increases from 100 to 200 nM to 600800 nM, which is slightly higher compared to the increase in cytosolic Ca 2+ , and gradually increases without the oscillatory nature (Wiederkehr et al., 2011)

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The pathogenicity and slow growth 3 to 4 weeks to form colonies make M

After ischemic brain injury, the oxidative stress pathway in the endoplasmic reticulum was reduced by inactivation of mitogen-activated protein kinases p38/MAPK and a decrease in the expression of the enhancer-binding proteins (C/EBP) and homologous protein (CHOP), leading to a reduction of inducible iNOS and nitrotyrosine production [87]

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