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Surprisingly, we now show genetic rescue by a single copy of the truncated C/EBP LIP isoform in fat development and homeostasis

During ischemia, overstimulation of NMDA receptors by glutamate and glycine triggers pathological calcium influx, leading to mitochondrial calcium overload and subsequent mitochondrial permeability transition pore (mPTP) opening, further initiating apoptotic cascades [144] (Fig

Nasal peptides are administered via the nasal cavity, and bypass the use of traditional injections, thus offering a painless, non-invasive alternative

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Furthermore, human Apo AI is known to be an effective inducer of endogenous Apo E production from lipid-loaded macrophages and independent of ABCA1-mediated cholesterol efflux, relating to NF-B inhibition and/or PKA pathway activation 115