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5.4 Enhancement of neutrophil phagocytic capacity Treatment of rats with acute myocardial infarction using the FPR2 agonist BMS-986235 enhances the phagocytic capacity of neutrophils, aiding in the clearance of dead cells and inflammatory mediators, lowering inflammatory damage, and enhancing adverse remodeling and cardiac function post-myocardial infarction (189)

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