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Further investigations are warranted to elucidate the precise role of mitochondria and unravel the underlying molecular mechanisms implicated in ferroptosis [138, 139]

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Moreover, while by using costaining with tumour markers or detecting tumour-specific proteins or transcripts, we showed that we are targeting senescent tumour cells

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In a BV-2 in vitro model, shifting from a high to normal glucose concentrations kept microglia in a proinflammatory and metabolically stressed state, with increased signs of autophagy (Hsieh et al., 2019)