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In a COPD mouse model, CS exposure led to endoplasmic reticulum stress, mitochondrial damage, and increased NLRP3 inflammasome formation

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Clinical tests show that GLP-1 peptide Cagrilintide keeps the metabolism stable even when dipeptidyl peptidase-IV and neutral endopeptidases break it down

MPH and amphetamine-derived compounds (AMPH), the two most widely used drugs for ADHD, act on DAT-1 by inhibiting its transporter function and thereby increasing the levels of extracellular DA [75]

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