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Perhaps counterintuitively, in some cancers glucose-6-phosphate may recycle back to glucose by first entering ER through the glucose-6-phosphate transporter where it is dephosphorylated by glucose-6-phosphatase and then transported as glucose to the cytoplasm through GLUTs on the ER membrane (Fig
Similar content being viewed by others Main Impaired mitochondrial oxidative phosphorylation (OXPHOS) generates excessive reactive oxygen species (ROS), leading to mitochondrial damage, oxidative stress and apoptotic cell death
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S2CID 72562308
Without heavy metal stress, gr knockout resulted in increased expression levels of glutathione synthetase and glutathione peroxidase, while gr overexpression resulted in increased expression levels of glutathione reductase, thioredoxin reductase, and heterodisulfide reductase subunit C and decreased expression levels of peroxiredoxin, glutathione synthetase, and glutathione S-transferase (Table 4)
Anti-angiogenic therapy in the treatment of non-small cell lung cancer
