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Autophosphorylation at tyrosine 397 is commonly used as a marker of FAK activation because it creates a binding site for Src-family kinases and supports downstream signaling events involved in adhesion turnover and migration-associated processes

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Pickart and colleagues reported that systemic (injectable) GHK-Cu induced wound healing in rats, mice, and pigs, suggesting that the peptide's effects extend beyond the local application site

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