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doi: 10.1177/1352458516652497 73 MahadDHTrappBDLassmannH

The healing of transected tendon, osteotendinous junction following Achilles tendon detachment, and myotendinous junction were reported [2,6,10]

Second, as an adaptive stress resistance mechanism, HIF-1 activates autophagy by upregulating BNIP3, facilitating the removal of damaged organelles such as mitochondria 94

This shift may be attributed to the previously documented gut microbiome alteration in FM subjects 15,38,39,40 or due to an increased intestinal permeability observed in FM subjects 41,42

demonstrated that HOTAIR binds to UPF1, which promotes the degradation of ACSL4, thereby reducing ferroptosis

Accordingly, the ability of colchicine to bind to free tubulin dimers, thus blocking microtubule polymerization and therefore TNF- secretion, is believed to be the key mechanism behind the broad spectrum of colchicine-mediated anti-inflammatory effects (96, 97)