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Likewise, the isoprenaline myocardial infarction was counteracted by NO-effect [38]

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Biphasic activation of apoptosis signal-regulating kinase 1-stress-activated protein kinase 1-c-Jun N-terminal protein kinase pathway is selectively mediated by Ca2 + -permeable -amino-3-hydroxy-5-methyl-4-isoxazolepropionate receptors involving oxidative stress following brain ischemia in rat hippocampus

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Markedly, multiple organ lesions in the heart, lung, liver, kidney, and gastrointestinal tract, in particular, as well as brain lesions, were attenuated, and oxidative stress was reduced in tissues (Vukojevic et al., 2018

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However, completely absent progress after 8-12 weeks suggests either ineffective protocol or injury severity beyond peptide intervention requiring medical evaluation

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Renal outcomes and all-cause death associated with sodium-glucose co-transporter-2 inhibitors versus other glucose-lowering drugs (CVD-REAL 3 Korea)

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