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Mol Psychiatry 2016

We also demonstrated that it had an unexpected influence on neighboring non-IgG + B cells

[16] [17] Whereas the latter directly act on the dopamine transporter (DAT) to inhibit the reuptake and/or induce the release of dopamine, bromantane instead acts via indirect genomic mechanisms to produce a rapid, pronounced, and long-lasting upregulation in a variety of brain regions of the expression of tyrosine hydroxylase (TH) and aromatic L-amino acid decarboxylase (AAAD) (also known as DOPA decarboxylase), key enzymes in the dopamine biosynthesis pathway

[DOI] [Google Scholar] 55.Bajoria P.S., Dave P.A., Rohit R.K., Tibrewal C., Modi N.S., Gandhi S.K., Patel P

Another challenge is to identify drugs discriminatively targeting synaptic and extrasynaptic NMDARs because they have different roles in brain function and in the pathogenesis of neuropsychiatric disorders [6]

Genome-wide analysis of histone modifications in human endometrial stromal cells