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moreover, the Gpx4 transgene may not be activated in this tissue context and suppression of the ferroptosis phenotype in vivo may require engineering of multiple ferroptosis regulatory genes

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Immunol Res (2017) 65(2):51223

While the specific details of the ECD interface of Cagri-AMY 3 R could not be determined due to the low-resolution maps, these findings are indicative of substantial loss of RAMP3/CTR ECD interface interactions and considerable dynamic motions within the RAMP3 and CTR ECD protomers (Fig

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