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It alleviates hepatic injury by activating the Nrf2/HO-1 antioxidant pathway and suppressing NF-Bmediated inflammation, highlighting its potential as a therapeutic applicant for ALD (94)

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It has been found that SiO 2 NPs can exacerbate PD pathology by promoting -synuclein aggregation, mitochondrial impairment, OS, and neuronal apoptosis

The first is via glutamate dehydrogenase (GLDH), which produces the potential autophagy inducer ammonium and NADH or NADPH as cofactors