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however, the formation of urolithiasis is a complex pathological process, and ferroptosis does not play the only role in the pathological process alone
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Although involvement of tetrodotoxin (TTX)-resistant Na V isoforms, most notably Na V 1.8 and Na V 1.9, in CIPN has been suggested (Descoeur et al., 2011
SOD2 is shown to have a unique genetic organization and little similarity with SOD1 and SOD3 [30]
