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It can be argued that the possible hypoglycemic effect is due to concomitant use of other therapies that cause hypoglycemia (eg, sulphonylurea) and it was demonstrated in clinical trials studying a combination of a GLP-1 receptor agonist and a sulphonylurea that the risk of hypoglycemia was higher when compared to placebo

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Elucidation of the physiology of endogenous GLP-1 was enabled by use of exendin(939), a selective antagonist of the mammalian GLP-1 receptor [40]

doi:10.1007/s00268-018-4479-z

Hoechst 33342, Annexin V, and PI were also from Thermo Fisher Scientific (Waltham, MA)

But supporting GLP-1 production and replicating what GLP-1 medications do are two very different things