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Secondary prophylaxis of hepatic encephalopathy in cirrhosis: an open-label, randomized controlled trial of lactulose, probiotics, and no therapy
Mitochondrial damage triggers the concerted degradation of negative regulators of neuronal autophagy
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This design minimises bias and provides the most reliable evidence of treatment efficacy
Determining the genegene and geneenvironment interactions are fundamental in epidemiological studies to identify the etiology of different diseases and predict the risk factors for disease prevention
In addition, TGS can significantly inhibit the expression of IL-1, IL-6 and NF-B, and enhance the expression of IL-10 in heart tissue (Figure 3C
