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The APOE-TR model is a mouse knock-in model where exons 24 (comprising the mouse APOE gene) are replaced with human APOE in 129xC57BL/6 mice back-crossed to C57BL/6 mice (Sullivan et al., 1997)

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Amorphous heads, two heads, merged body, two tails, and a regularly formed head with a broken or twisted tail were all considered abnormal

Furthermore, lipid systems can be engineered into vesicular structures to improve flexibility

Antioxidant defense and oxidative damage vary widely among high-altitude residents