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Description
Both in vivo studies in mice and in vitro studies in cultured human aortic endothelial and vascular smooth muscle cells have shown that physiological levels of TMAO induce expression of inflammatory cytokines and adhesion molecules
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Argininosuccinate lyase deficiency
Notably, defects in mitophagy also were reported at the onset and progression of neurodegenerative diseases
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Medium-chain and short-chain fatty acids that are not broken down can accumulate in tissues and damage the liver, heart and muscles, causing serious complications
