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Oxidative phosphorylation is disrupted at complex I and II in GSH-deficient follicular B cells To determine whether the accumulation of succinate in Gclc -deficient FoB was caused by a dysfunctional ETC, we assessed the respiratory parameters of each ETC complex
A., Chiang, J
Gojznikar, B
A transcriptomic analysis of cerebral microvessels reveals the involvement of Notch1 signaling in endothelial mitochondrial-dysfunction-dependent BBB disruption
C.VincentB
Another study also proved a significant increase in ROS production and protein carbonylation together with a decrement of antioxidants in skeletal muscle of NFE2L2-/- mice
