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Epileptogenic injuries or seizures initiate the synthesis and secretion of proinflammatory molecules such as TNF-, IL-1, and High Mobility Group Box 1 (HMGB1) in glial cells which result in decreasing seizure threshold, which further contributes to seizure precipitation, and recurrence because of rapid changes in glutamate and -aminobutyric acid (GABA) receptor phosphorylation
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In addition, the existing NOX inhibitors are mainly obtained by high-throughput screening
The results showed that FA injection markedly enhanced the secretion of TNF- and IL-1 in the tubular epithelium, together with the increased infiltration of macrophage in the interstitial space, whereas LA supplementation downregulated the levels of these molecules
Vora LK, Gholap AD, Jetha K, Thakur RRS, Solanki HK, Chavda VP
J., Cherry, C
