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doi:10.1038/nn.2146
8 , ofab389 (2021)
Lupus autoantibodies initiate neuroinflammation sustained by continuous HMGB1:RAGE signaling and reversed by increased LAIR-1 expression
In renal disorders, excessive mitochondrial ROS together with ROS derived from NOX enzymes intensify mitochondrial injury and dissipate the organelles membrane potential (40, 41)
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doi: 10.1002/mas.20207
