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In hepatocytes, p38 can either exacerbate or protect against toxin-induced cell death depending on the specific isoform (p38, p38, p38, p38) and the timing of activation

Another cytosolic protein translocating to the mitochondria after APAP is the glycogen synthase kinase-3 (GSK-3), which is a major regulator of glycogen synthase but has been shown to regulate other processes, including cell death65

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Read more about the full framework at The Solensis Science Framework

This is the opposite of the antagonistic relationship seen with ibuprofen

1D,E, and bar graphs in Fig