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Collectively, our results suggest that upregulation of the glutamate transporter EAAT3 is an adaptation of EBV-infected cells to maintain cellular redox homeostasis against the virus-induced oxidative stress, and that this cellular balance could be therapeutically destroyed by targeting EAAT3 to impede EBV-associated cancers.

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Under physiological conditions, reduced glutathione accounts for the vast majority

Algae exposed to stress from the very beginning were more susceptible to HM ions than grown cultures