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Briefly, the liver tissue specimens were sequentially fixed with 2.5% glutaraldehyde and 1% osmium tetroxide

[DOI] [PMC free article] [PubMed] [Google Scholar] FDA, EMA, Huebener P., Pradere J

In addition, dichloroaceetate treatment alone caused ER stress to an extent that was nearly as robust as the bona fide ER stressor TM, as seen by upregulation of UPR target genes (Figure 7B) and splicing of Xbp1 (Figure 7C)

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It functions similarly to CJC-1295 (without DAC), promoting physiologic GH pulses that support recovery, tissue repair, and systemic regeneration

doi: 10.1152/ajpgi.1999.276.5.G1279