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Mechanistically, augmented ischemia-reperfusion injury in the absence of GPx-1 may be caused by increased NFB activation, as pyrrolidine dithiocarbamate (PDTC), an NFB inhibitor, partially protects against neurotoxicity in this model (82)

doi: 10.1007/s12033-011-9458-7

Patients tracking their progress with before and after documentation should take body composition measurements (not just scale weight) to capture the glycine protective effect on lean mass

For lupus researchers, the LL-37 story provides important lessons

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Clinical and demographic predictors of long-term disability in patients with relapsing-remitting multiple sclerosis: a systematic review
