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One such facet to malaria parasites is their ability to extensively remodel host cells by exporting numerous parasite proteins 15,16,17,18

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This effect may be mediated, at least in part, by an increase in neurotrophic factors and acetylcholinesterase-inhibiting activity, as well as a reduction in amyloid burden and tau pathology in the cerebral cortex

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Plasma homocysteine and severity of atherosclerosis in young patients with lower-limb atherosclerotic disease

HMGCS1 drives drug-resistance in acute myeloid leukemia through endoplasmic reticulum-UPR-mitochondria axis