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Specification: Brand: HRB

It has been shown, that if the core protein is mutated, the association with LDs might be disrupted

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A fourteen-day continuous intraventricular infusion of MT-II in normal C57BL/6J mice did not induce any significant changes in sociability or social novelty scores compared to vehicle-treated controls

Summary Atherosclerotic lesions initiate with endothelial cell dysfunction causing modification of apoB containing lipoproteins (LDL, VLDL, remnants) and infiltration of immune cells, particularly monocytes, into the subendothelial space (Figure 1)