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J., Alam, J

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213 p62 and dipeptidyl peptidase 9 (DPP9) disrupt the interaction between KEAP1 and NRF2 by competitively binding to KEAP1 to maintain the NRF2 stability and promote the transcription of its downstream iron metabolism and antioxidant genes, resulting in ferroptosis-mediated sorafenib resistance

Bioinformatics and tissue analyses revealed elevated SLC25A39 protein levels in LUAD despite stable mRNA expression, with higher protein expression correlating with poorer patient survival