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doi: 10.1016/j.rechem.2024.101463 209 QiX.ZhuK.KeW.WangJ.MaoS.ChenG

Arch Neurol 36: 6573, 1979

doi: 10.1097/TP.0000000000002882 64 YaoQHuangZWZhaiYYYueMLuoLZXuePPet al

Nitric oxide scavenging by red blood cell microparticles and cell-free hemoglobin as a mechanism for the red cell storage lesion

Following ph-IOP-induced damage, it effectively binds to excess divalent iron in retinal tissue, preventing toxic accumulation

pretreated for 1 h) significantly attenuates the uridine 5-triphosphate (UTP)-induced increases of [Ca 2+ ] i in A431 cells [3]
