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& Blendy, J.A
The SP1/ACSL4 pathway has been shown to be associated with ferroptosis and may serve as a potential therapeutic target in mouse models of intestinal ischemia necrosis and Alzheimer's disease [57, 58]
It can reduce the serum level of inflammatory cytokines, such as IL-2, interferon-gamma, subunit beta of interleukin 12 (IL-12p40), and monocyte chemoattractant protein-1 (MCP-1) (Fig
Axonal transport can be disrupted through various mechanisms, including damage to motor proteins and cytoplasmic dynein, microtubules, cargo, and mitochondria [42, 43]
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