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While complement deposition is still understood as a central mechanism, it was recently demonstrated that induction of optic neuritis by human AQP4ab in mice is dependent on Interferon type I signaling (94)

Three decades later, translational research has further developed procedures, including direct activation of immune-effector cells against specific targets in an MHC-independent manner (2, 3)

The characterization of the Caenorhabditis elegans mitochondrial thioredoxin system uncovers an unexpected protective role of thioredoxin reductase 2 in beta-amyloid peptide toxicity

doi: 10.3389/fonc.2020.578315

Methotrexate (MTX) is the most commonly used (first-line) DMARD

It is anticipated that as the population ages, the incidence of AD will increase, placing a greater strain on healthcare systems worldwide