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Here we briefly review the evidence showing that changes to oligodendrocytes and myelin are key in neurodegeneration, and explore the relevance of DNA methylation in oligodendrocyte (dys)function

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It preserves cellular damage recovery, boosts collagen production, and maintains telomere length

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We model the reaction as linear and reversible because covalent binding of NAPQI gradually declines after eight hours[28, 29]