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This endothelial dysfunction contributes to the onset and advancement of diabetic vascular complications, such as coronary artery disease (macrovascular) and retinopathy (microvascular) [39]

This suggests that it may be most effective in combination with other agents

Oxidative stress can disrupt these tolerance mechanisms by inducing apoptosis of regulatory T cells (Tregs), which are critical for maintaining immune homeostasis and preventing autoimmunity [141]

Across hundreds of animal studies, serious adverse effects have not been commonly reported

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(2010) observed L5 of the barrel cortex and concluded likewise, that Fmr1 knockout mice have a larger pool of unstable spines which accounts for the increased dynamics