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Description
JAMA 1972
19 also received their urine samples from the MAPP Research Network, but completed their study approximately 6-years prior to ours
This study was key to establishing the idea that abnormal excitability of motor-system neural function could be a physiological mechanism underlying dystonia
doi: 10.1289/ehp.1408524
(2020) 52:e13816
TLR7) through XIST-dependent histone deacetylation, since these genes lack promoter DNA methylation [53]
