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J Neurochem 75:21782189

This balance influences numerous redox-regulated cellular processes, including the modulation of signaling pathways, post-translational modifications such as protein glutathionylation, activation of transcription factors like Nrf2 and NF-B, and the regulation of cell fate decisions such as proliferation or apoptosis [38]

A key open question is how human and microbial enzymes work together to degrade AGEs and maintain their concentrations within a potentially beneficial range

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Consumption of redox energy by glutathione metabolism contributes to hypoxia/reoxygenation-induced injury in astrocytes

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