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They disrupt bacterial cell membranes and inhibit biofilm formation, making them effective against pathogens such as Escherichia coli and Staphylococcus aureus [25]

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Hoke and colleagues [72] demonstrated a marked upregulation in both mRNA expression and protein concentrations of key components within the PI3K/AKT/mTOR signaling cascade during different phases of cutaneous wound repair in murine models