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Discussion We propose that, in order to prevent PD with high levels of efficacy and safety, mitochondrial nutrients should be combined, and they should be combined at optimal doses by targeting key features of the disease, i.e

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Readmission and mortality in malnourished, older, hospitalized adults treated with a specialized oral nutritional supplement: a randomized clinical trial

Intestinal absorption of carnosine and its constituent amino acids in man

356 Targeting negative regulators of p53 The above drugs are not applicable to cancers expressing wild-type p53

Abnormal tau, mitochondrial dysfunction, impaired axonal transport of mitochondria, and synaptic deprivation in Alzheimers disease