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A deficiency in either vitamin can impair homocysteine remethylation, highlighting the interdependence between folate and B12 for optimal metabolic function

The resulting vasogenic edema, inflammation, and oxidative stress further exacerbate neuronal damage and hinder recovery

Role of lectin-like oxidized low-density lipoprotein receptor-1 in inflammation and pathogen-associated interactions

Flippase malfunction, either from oxidative stress, caspase cleavage, or genetic lack, disturbs asymmetry and fosters hemolytic abnormalities

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10.1016/j.lf10.1016/j.lfs.2011.01.015 37 IlicS.DrmicD.ZarkovicK.KolencD.CoricM.BrcicL.et al (2010)