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3.3.3 Impact of GPX4 on immune-synovial interactions and joint microenvironment The contribution of GPX4 and ferroptosis to OA pathology extends far beyond direct protection of chondrocytes

Additionally, GO@GSH-FA nanoparticles displayed a visible corona structure around GO core, further confirming successful functionalization with the fibrinogen aptamer

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Ashraf et al., Labile Iron Pool and Its Role in Ferroptosis Increased LIP can lead to the generation of reactive oxygen species via Fenton reaction, eventually resulting in catastrophic membrane rupture (Kakhlon and Cabantchik, 2002

Previous studies have associated serotonin downregulation with schizophrenia (Shah and Gonzlez-Maeso, 2019), and postmortem analyses of brain tissue from schizophrenia patients revealed significantly elevated methylation levels at the HTR2A promoter region ( MB-COMT and 5-HT2A was partially corrected by antipsychotic treatment, and hypermethylation of 5-HT2A in the prefrontal cortex was correlated with treatment response (Figure 6E)