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What they found (summary): Amylin is a critical partner of insulin in metabolism, the loss of amylin in T2DM is as significant as the loss of insulin The AMY1 receptor in the area postrema is the primary target for the appetite effect Amyloid aggregation is the main problem with native amylin, proline substitutions (the Pramlintide strategy) are essential The half-life of native amylin is only 13 minutes, that is why lipidated analogs like Cagrilintide are necessary Perspective: amylin + GLP-1 combinations are the natural evolution of metabolic pharmacotherapy Why it matters: This is the reference article for amylin pharmacology

2.3.3 Neurological inflammation loop Neuroinflammation is a significant indicator of PD and plays a pivotal role in its pathogenesis and progression (Kubota et al., 2020)

Occult stage IIIA-N2 patients have excellent overall survival with initial surgery

Preventive medications include medicines for blood pressure control, antidepressants, anti-seizure drugs, botox injections, and CGRP monoclonal antibodies

Mitochondria-targeted hydrogen sulfide donors versus acute oxidative gastric mucosal injury

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