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The methioninecholine-deficient diet resulted in increased pro-inflammatory markers (IL-1b, tumor necrosis factor alpha (TNF ), and IL-6), increased ROS as well as increased lipid accumulation in mice [85]
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These increase in the levels will lead to oxygen consumption of lung mucus, transforming the lung microenvironment from aerobic to anaerobic, resulting in an increase in anaerobic bacteria
SEL is an antagonist of XOP1
Long-term outcomes in nephropathic cystinosis: a review
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