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Since the liver is the major site of IGF-1 production, when steatosis develops lowering insulin sensitivity, the severity of steatosis at different stages of insulin resistance and metabolic syndrome seems to be correlated with worsened circulating IGF-1 levels [234]

In the absence of Smad3, myostatin induces ROS production through the activation of p38 and ERK mitogen-activated protein kinase (MAPK) pathways mediated via TNF- , IL-6, NOX and XO [35]

The upregulation of autophagy levels can cause the overexpres-sion of the NCOA4 gene, and NCOA4 can cause iron storage as a cargo receptor

see Table 1)

At the same time, this complex organ ensures the maternofetal exchange of molecules, including those originating from the maternal microbiota (55)

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