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In BM-MSCs, METTL14 regulated osteogenic differentiation via the mA/IGF2BPs/Beclin-1 signaling axis [126], while METTL16 suppressed osteogenesis by promoting mA modification of PPAR transcripts, thereby enhancing its expression [127]

Nephroprotective effects of N-acetylcysteine amide against contrast-induced nephropathy through upregulating thioredoxin-1, inhibiting ASK1/p38MAPK pathway, and suppressing oxidative stress and apoptosis in rats

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6th International Stockholm/Sprinfield symposium on advances in Alzheimer Therapy
