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Their pathogenesis typically results from the long-term accumulation of factors such as pathological protein aggregation and disrupted MQC
The ANT crystal structures in both cytoplasmic-open (c-state) and matrix-open states (m-state) locked by two inhibitors bound at the solute binding site, carboxyatractyloside and bongkrekic acid respectively, highlighted a conserved alternating-access transport mechanism 5 enabled by the conformational change upon solute binding at the central cavity 6,7
The brain receiving less glucose
It is common for people with B12 deficiency anemia to have glossitis and stomatitis
