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The Expression of Nicotinamide N-Methyltransferase Increases ATP Synthesis and Protects SH-SY5Y Neuroblastoma Cells Against the Toxicity of Complex I Inhibitors

Mitochondrial dysfunction can drive senescence via reactive oxygen species (ROS) accumulation [68, 69], and Tat ( morphine) perturbs mitochondrial function, increasing ROS, protein oxidation, and cytokine release in microglia [60, 70]

Left atrial enlargement resulting from obesity was identified as the primary mediator of AF, underscoring a significant correlation between obesity and atrial structural remodeling (Parish, 2005)

[DOI] [PMC free article] [PubMed] [Google Scholar] 337.Liu J., Zhang C., Wang J., Hu W., Feng Z

Primary Handling Editor: Jennifer Sargent, in collaboration with the Nature Medicine team