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Ann Neurol 2013

Overall, the underlying pathophysiological mechanism of bone fragility in diabetes is very complex, including hyperglycemia, oxidative stress, and the accumulation of advanced glycosylation end products, which will damage the characteristics of collagen, increase bone marrow obesity, release inflammatory factors and adipokines from visceral fat, and may change the function of bone cells
In this context, persister cells may not be passive survivors but may actively adopt transcriptional and epigenetic states promoting immune evasion, a feature that can be observed across multiple tumor types but not yet fully established for persister populations

Miyamoto Y, Hurysz BM, Fajtova P, Eckmann P, Serafim MSM, Kronenberger T, et al

The Role of Selenium in Enhancing Glutathione Activity Selenium is a trace mineral that is crucial for numerous physiological processes in the body
Adults are less likely to experience this, but its still important to be aware
