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The amplification of MUC1 gene led to over-expression of the MUC1 C-terminal subunit (MUC1-C) complex, which could mediate the TNBC cell self-renewal ability and tumor origin [373]

doi: 10.3390/nu13030886 24 UllahHArbabSTianYChenYLiu C-qLQLiKa

Some of the upregulated pathways in ARIH2 and CCNC KO NK cells were cytokine receptor interaction, IFN response, and IL signaling pathways

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if the serum Mg2+ concentration is below 0.7 mM, it is hypomagnesemia