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PQQ also protects neurons by preventing the long-term overactivation of NMDA receptors, which results in excitotoxicity
European Neurology, 27 (2), 120129.
Currently, no enzyme has been shown to serve as a catalyst of S -glutathionylation in situ , although roles for human glutaredoxin-1 (GRX1
[PubMed: 9661618] 227
Teskey, G., et al., Glutathione as a Marker for Human Disease
The increased level of lipid peroxidation was positively correlated with severity of the disease and organ damage especially in nephritis patients [5053]
