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[DOI] [PubMed] [Google Scholar] 5.Kaplan EN
Although these mechanisms are part of normal healing, excessive or prolonged inflammation can cause serious complications (Jakob, 2001)
Puskarich MA, Kline JA, Krabill V, Claremont H, Jones AE
KCNQ2/3 openers show differential selectivity and site of action across multiple KCNQ channels
These results indicate that the activation of endogenous PLC-3 by -toxin contributes to increased production of G-CSF from endothelial cells, but it is independent of JNK signaling pathway
10.1093/ajcn/76.5.961 133 SchwahnB
