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Identification of protor as a novel rictor-binding component of mTOR complex-2

Recent experimental studies demonstrate that stabilizing mast cells with cromolyn after ischemic stroke reduces peripheral and central inflammation, preserves gut barrier integrity, mitigates dysbiosis, and improves functional outcomes, underscoring the pivotal role of gut-derived mast cells and histamine in post-stroke neuroinflammation (Conesa et al., 2023)

SETDB1 deficiency clearly promoted the acquisition of the TGF--induced mesenchymal phenotype but reduced the expression of E-cadherin in human alveolar epithelial cells by catalyzing the deposition of H3K9me3 on Snai1 , which inhibited the protein expression of Snai1, the main transcription factor that initiates the epithelial-mesenchymal transition (EMT)

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Pesticide Biochem Physiol 181:104996