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Upon restoration of normal levels of free chaperones, Hsfs are inactivated by binding of the HSP70 machinery [64, 74]

Similarly, Baker and team affirmed that preexisting Toxoplasma gondii infection augments brain damage during TBI pathogenesis by modulating glutamatergic, neurotoxic, and oxidative stress markers, along with significant elevation in the expression of neuroinflammatory mediators [324]
PMID: 27895400 Citations are provided for research reference

doi:10.1016/j.atherosclerosis.2014.04.004 Moser MA, Chun OK

Accessed March 2, 2008 2

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